Oxprenolol is a lipophilic, nonselective beta-adrenergic receptor antagonist with anti-arrhythmic, anti-anginal and antihypertensive activities. Oxprenolol competitively binds to and blocks beta-1 adrenergic receptors in the heart, thereby decreasing cardiac contractility and rate. This leads to a reduction in cardiac output and lowers blood pressure. In addition, oxprenolol prevents the release of renin, a hormone secreted by the kidneys that causes constriction of blood vessels.

Butofilolol

MSDS

CAS:64552-17-6

Molecular Formula:C17H26FNO3

Alias

More Information

1-[2-[3-(Tert-Butylamino)-2-Hydroxypropoxy]-5-Fluorophenyl]Butan-1-One; Cafide

Brief Introduction

Butofilolol (trade name Cafide) is a beta-blocker drug for the treatment of essential hypertension (high blood pressure). It is not known to be marketed anywhere.

Bepridil

CAS:64706-54-3

Molecular Formula:C24H34N2O

Alias

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Vascor; Cordium; Bepadin (Tn); Cerm 1978; Org 5730; Bepadin; N-Benzyl-N-[3-(2-Methylpropoxy)-2-Pyrrolidin-1-Ylpropyl]Aniline; Bepridil (Inn)

Brief Introduction

New calcium antagonists can selectively dilate coronary arteries in order to inhibit calcium slow channels. In addition, they can also inhibit sodium and potassium channels. Therefore, they have the characteristics of class IA, III and IV antiarrhythmic drugs. The half-life is up to 42h. It can be administered once a day. It was well tolerated and had less toxic and side effects. It is mainly used for the prevention and treatment of angina pectoris, arrhythmia, hypertension and coronary heart disease.

Bis(Sulfanylidene)Molybdenum,2-Hydroxyethyl(Trimethyl)Azanium,Sulfanide

MSDS

CAS:649749-10-0

Molecular Formula:C10H30MoN2O2S4

Alias

More Information

Bis(Choline)Tetrathiomolybdate; Unii-Fd57A79R4P; Ethanaminium,2-Hydroxy-N,N,N-Trimethyl-,(T-4)-Tetrathioxomolybdate(2-)(2:1); Atn 224

Brief Introduction

Tiomolibdate Choline is an orally active second generation tetrathiomolybdate analog with anti-angiogenic and antineoplastic activities. Tiomolibdate choline selectively chelates the copper ion in superoxide dismutase 1 (SOD1) in endothelial cells, thereby depleting SOD1 of copper and inhibiting its activity. Inhibition of SOD1 interferes with the activation of several signal transduction pathways required for cellular proliferation and angiogenesis, including those mediated by ERK1/2 and FAK and Src kinases. This results in an inhibition of cell proliferation and angiogenesis as well as induction of apoptosis.

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